Escherichia coli K-12 gyrB gene product is involved in the lethal effect of the ligts2 mutant of bacteriophage Mu.
AUTOR(ES)
Ghelardini, P
RESUMO
Mu ligts2 mutants, defective for development and integration, show a high killing effect on the infected host. A number of survivors to Mu ligts2 infection were analyzed; they are characterized by nonpermissivity for both development and lysogenization of bacteriophage Mu. Bacteriophages D108 and P1 are also inhibited in these strains as is transposon Tn9. The corresponding mutation site was mapped at 82 min and identified with the Escherichia coli gyrB site.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=215300Documentos Relacionados
- Involvement of Escherichia coli K-12 DNA polymerase I in the growth of bacteriophage Mu.
- Gene expression in a temperature-sensitive gyrB mutant of Escherichia coli.
- Quinolone resistance-determining region in the DNA gyrase gyrB gene of Escherichia coli.
- gyrA and gyrB mutations in quinolone-resistant strains of Escherichia coli.
- Escherichia coli K-12 and B contain functional bacteriophage P2 ogr genes.