Genetic Deficiency of Itgb2 or ItgaL Prevents Autoimmune Diabetes Through Distinctly Different Mechanisms in NOD/LtJ Mice
AUTOR(ES)
Glawe, John D.
FONTE
American Diabetes Association
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2682677Documentos Relacionados
- Inhibition of Th17 Cells Regulates Autoimmune Diabetes in NOD Mice
- Beta cell expression of endogenous xenotropic retrovirus distinguishes diabetes-susceptible NOD/Lt from resistant NON/Lt mice.
- Metabolically inactive insulin analog prevents type I diabetes in prediabetic NOD mice.
- Targeting B-cells Mitigates Autoimmune Diabetes in NOD Mice: What Is Plan B?
- Two novel frame shift, recurrent and de novo mutations in the ITGB2 (CD18) gene causing leukocyte adhesion deficiency in a highly inbred North African population