Invasive Lesions Containing Filamentous Forms Produced by a Candida albicans Mutant That Is Defective in Filamentous Growth in Culture
AUTOR(ES)
Riggle, Perry J.
FONTE
American Society for Microbiology
RESUMO
A Candida albicans efg1 cph1 double mutant is nonfilamentous under standard laboratory conditions and avirulent in mice. However, this mutant produced filaments in the tongues of immunosuppressed gnotobiotic piglets and when embedded in agar, demonstrating that an Efg1p- and Cph1p-independent pathway for promotion of filamentous growth exists.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=116558Documentos Relacionados
- Lack of Catheter Infection by the efg1/efg1 cph1/cph1 Double-Null Mutant, a Candida albicans Strain That Is Defective in Filamentous Growth
- Candida albicans Septin Mutants Are Defective for Invasive Growth and Virulence
- A G1 Cyclin Is Necessary for Maintenance of Filamentous Growth in Candida albicans
- VIRULENCE ENHANCEMENT OF A FILAMENTOUS STRAIN OF CANDIDA ALBICANS AFTER GROWTH ON MEDIA CONTAINING CYSTEINE
- Defective Induction of Interleukin-12 in Human Monocytes by Germ-Tube Forms of Candida albicans
