Mechanism of Natriuresis after Closure of Chronic Arteriovenous Shunts
AUTOR(ES)
Mandin, Henry
RESUMO
Animals subjected to certain cardiovascular manipulations, such as arteriovenous fistulas, diminish their urinary sodium excretion. It has been shown that closure of such fistulas results in a prompt increase in the rate of sodium excretion. However, the nature of the renal mechanisms increasing the excretion of sodium when the initial cardiovascular abnormality is corrected has remained unclear. Since the elucidation of such mechanisms might provide information pertinent to other sodium-retaining states, the effect of closure of chronic Teflon-Silastic arteriovenous shunts was studied in desoxycorticosterone acetate (DOCA)-treated dogs by utilizing micropuncture techniques.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=333024Documentos Relacionados
- Subdural haematoma after the treatment of chronic hydrocephalus by ventriculocaval shunts.
- The Mechanism of Denervation Natriuresis *
- The mechanism of the natriuresis of fasting.
- Closure of Blalock-Taussig shunts using a new detachable coil device.
- Arteriovenous shunts in the peropheral pulmonary circulation in the human lung.