Simian virus 40 small-t antigen-induced theophylline resistance is not mediated by cyclic AMP.
AUTOR(ES)
Renz, C
RESUMO
Small-t antigen of simian virus 40 renders CV-1 cells resistant to growth arrest induced by theophylline and other methylxanthines. Elevated levels of cyclic AMP are not involved in growth arrest of CV-1 cells by methylxanthines, and small-t antigen does not alter cyclic AMP levels dramatically after infection.
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=254879Documentos Relacionados
- Mutational analysis of simian virus 40 small-t antigen.
- Simian virus 40 small-t antigen binds two zinc ions.
- Control of protein phosphatase 2A by simian virus 40 small-t antigen.
- A novel simian virus 40 early-region domain mediates transactivation of the cyclin A promoter by small-t antigen and is required for transformation in small-t antigen-dependent assays.
- Reversal of simian virus 40 small-t-antigen-induced theophylline resistance.