The C3a Anaphylatoxin Receptor Is a Key Mediator of Insulin Resistance and Functions by Modulating Adipose Tissue Macrophage Infiltration and Activation
AUTOR(ES)
Mamane, Yaël
FONTE
American Diabetes Association
ACESSO AO ARTIGO
http://www.pubmedcentral.nih.gov/articlerender.fcgi?artid=2731537Documentos Relacionados
- Anaphylatoxin C3a receptors in asthma
- Adipose Tissue Exosome-Like Vesicles Mediate Activation of Macrophage-Induced Insulin Resistance
- Cardiac dysfunction caused by purified human C3a anaphylatoxin.
- Adipocyte macrophage colony-stimulating factor is a mediator of adipose tissue growth.
- Synthetic peptides with the biological activities and specificity of human C3a anaphylatoxin.
