Benign Prostatic Etiology
Mostrando 1-7 de 7 artigos, teses e dissertações.
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1. Lack of evidence of HPV etiology of prostate cancer following radical surgery and higher frequency of the Arg/Pro genotype in turkish men with prostate cancer
ABSTRACT Objectives The aim of this study was to assess the possible role of HPV in the development of prostate cancer (PCa) and investigate the distribution of the p53 codon 72 polymorphism in PCa in a Turkish population. Materials and methods A total of 96 tissues, which had been obtained using a radical surgery method, formalin-fixed and parafin-embed
Int. braz j urol.. Publicado em: 2017-02
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2. O papel dos micros RNAs 143 e 145 e seus genes alvo na etiopatogenia da hiperplasia prostática benigna / The role of micro RNAs 143 and 145 and their target genes in the etiopathogenesis of benign prostatic hyperplasia
Introdução: A hiperplasia prostática benigna (HPB) é apontada como um dos mais comuns problemas de saúde associado ao envelhecimento dos homens. A incidência da doença começa a aumentar a partir dos 40 anos de idade, e se torna frequente em cerca de 50% dos homens com 50 anos e em quase 90% dos homens após a oitava década. A etiopatogenia da HPB ai
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 04/12/2012
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3. Efeito inibidor da finasterida sobre a enzima 5-alfa-redutase na prostata de gerbilos adultos (Meriones unguiculatus)
Prostatic deve1opment and differentiatíon are induced by androgens acting via mesenchymal-epithelial interactions, and during adult life the gland is maintained functionally active by reciprocal homeostatic stromal-epithelial interactions. Hormonal alterations, usually associated with the physical aspects of aging, leads to disruptions of this homeostatic i
Publicado em: 2003
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4. Symptomatic and asymptomatic benign prostatic hyperplasia: Molecular differentiation by using microarrays
Benign prostatic hyperplasia (BPH) is a disease of unknown etiology that significantly affects the quality of life in aging men. Histologic BPH may present itself either as symptomatic or asymptomatic in nature. To elucidate the molecular differences underlying BPH, gene expression profiles from the prostate transition zone tissue have been analyzed by using
The National Academy of Sciences.
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5. Tissue content of dihydrotestosterone in human prostatic hyperplasis is not supranormal.
The dihydrotestosterone content of normal peripheral and benign hyperplastic prostates was measured in tissue obtained at open surgical procedures on 29 men of ages 36 to 82 yr. The dihydrotestosterone content in normal prostates (mean +/- SE, 5.1 +/- 0.4 ng/g tissue) and in benign hyperplastic prostates (5.0 +/- 0.4) was similar. In 11 patients in whom both
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6. Detection of human papillomavirus DNA in prostate gland tissue by using the polymerase chain reaction amplification assay.
Human papillomavirus (HPV) is associated with specific benign and malignant lesions of the epithelial and mucosal surfaces. Of the sexually transmitted types, HPV type 16 (HPV 16) and HPV 18 are commonly associated with severe dysplasia and carcinoma of the uterine cervix. In men, genital HPV infections which have been studied are manifest as external lesion
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7. A Signaling Network in Phenylephrine-Induced Benign Prostatic Hyperplasia
Benign prostatic hyperplasia (BPH) is an age-related disease of unknown etiology characterized by prostatic enlargement and coinciding with distinctive alterations in tissue histomorphology. To identify the molecular mechanisms underlying the development of BPH, we conducted a DNA microarray study using a previously described animal model in which chronic α
The Endocrine Society.