Hormones Of The Renal System
Mostrando 1-12 de 14 artigos, teses e dissertações.
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1. Gender-dependent effects of aging on the kidney
It is well known that the kidney plays an important role in the development of cardiovascular diseases such as hypertension. The normal aging process leads to changes in kidney morphology, hemodynamics and function, which increase the incidence of cardiovascular events in the elderly population. These disturbances are influenced by several factors, including
Brazilian Journal of Medical and Biological Research. Publicado em: 2011-09
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2. Efeitos não-genômicos dos hormônios esteróides - aldosterona e corticosterona - sobre a acidificação do túbulo proximal (S2) de ratos: estudos de microperfusão tubular e capilar, in vivo . / Nongenomic effect of steroid hormones - aldosterone and corticosterone - on acidification of rat proximal tubule (S2) studies by tubular and capillary microperfusion, in vivo .
The purpose was to determine if aldosterone and corticosterone act on the acidification of proximal tubule and if these hormonal effects are genomic and/or nongenomic. Bicarbonate reabsorption was evaluated by microperfusion. Aldosterone and corticosterone caused a significant increase in JHCO3-. In the presence of ethanol, actinomycin D, cycloheximide or es
Publicado em: 2010
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3. Stereotactic radiosurgery in the treatment of brain metastases: efficacy and complications based on brain location / Radiocirurgia no tratamento das metástases cerebrais: resposta terapêutica e complicações baseadas na localização da lesão
Brain metastases are the most common Central Nervous System tumors. Among the therapy options stereotactic radiosurgery has became in the last two decades an usefulness treatment technique attending with lower complication and mortality rate. However, even an extended literature review, many questions remain unclear about this therapy modality, mainly relate
Publicado em: 2006
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4. Nitrergic modulation of vasopressin, oxytocin and atrial natriuretic peptide secretion in response to sodium intake and hypertonic blood volume expansion
The central nervous system plays an important role in the control of renal sodium excretion. We present here a brief review of physiologic regulation of hydromineral balance and discuss recent results from our laboratory that focus on the participation of nitrergic, vasopressinergic, and oxytocinergic systems in the regulation of water and sodium excretion u
Brazilian Journal of Medical and Biological Research. Publicado em: 2002-09
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5. Neurohumoral mechanisms and the role of arterial baroreceptors in the reno-vascular response to haemorrhage in rabbits.
1. Conscious rabbits, with implanted renal artery Doppler flow probes were bled at a constant rate (4 ml min-1). We assessed the contribution of autonomic, hormonal and local factors to the renal vasoconstrictor response to 20% loss of blood volume (BV) and the role of the sinoaortic baroreceptors in the neurohumoral response. 2. With intact autonomic effect
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6. Neurohypophyseal Hormone-Responsive Adenylate Cyclase from Mammalian Kidney
The investigation was undertaken to evaluate the direct stimulatory effects of neurohypophyseal hormones upon adenylate cyclase activity in a cell-free, particulate fraction derived from the kidney medulla of various mammalian species. The relative affinity of neurohypophyseal hormones for the receptor component of the adenylate cyclase system (as defined by
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7. Increased renal secretion of norepinephrine and prostaglandin E2 during sodium depletion in the dog.
To determine whether vasoactive renal hormones modulate renal blood flow during alterations of sodium balance, simultaneous measurements of arterial and renal venous concentrations of norepinephrine and prostaglandin E2 (PGE2) and of plasma renin activity, as well as renal blood flow and systemic hemodynamics were carried out in 24 sodium-depleted and 28 sod
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8. Mechanism of resistance to the phosphaturic effect of the parathyroid hormone in the hamster.
The effect of parathyroid hormone and calcitonin on the renal excretion of phosphate, calcium, and cyclic AMP was evaluated in the thyroparathyroidectomized hamster, a mammal apparently reisstant to the phosphaturic effect of parathyroid hormone. Parathyroid hormone did not increase phosphate excretion, although it decreased excretion of calcium and increase
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9. Nitric oxide, cGMP, and hormone regulation of active sodium transport.
The inter- and intracellular regulator nitric oxide (NO) has been suggested to play a role in the modulation of cellular excitability, but the mechanism(s) by which this occurs remain unclear. Using the kidney as a model system, we report here evidence that NO, produced in response to various hormones and cytokines, can effect long-term alterations in the ac
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10. Thyrocalcitonin, EGTA, and Urinary Electrolyte Excretion*
The infusion of thyrocalcitonin (TCT) into thyroparathyroidectomized rats, given either no exogenous parathyroid hormone or a constant infusion of this hormone, leads to a transient phosphaturia and a decreased excretion of urinary magnesium, calcium, and hydroxyproline without a change in glomerular filtration rate. The changes in phosphate excretion may be
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11. Sodium-independent in vitro induction of Na+,K+-ATPase by aldosterone in renal target cells: permissive effect of triiodothyronine.
The aim of this study was to develop an in vitro system in which we could study the causal relationship between short-term stimulation of Na+,K+-ATPase in the collecting tubule by aldosterone on the one hand and protein synthesis and changes in intracellular Na+ concentration on the other hand. Previous in vivo studies suggested that triiodothyronine might f
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12. Estrogen up-regulates Bcl-2 and blocks tolerance induction of naïve B cells
Sex hormones are presumed to contribute to sexual dimorphism in the immune system. Estrogen, in particular, has been suggested to predispose women to systemic lupus erythematosus. We report here that estradiol (E2) can break B cell tolerance and induce a lupus-like phenotype in nonautoimmune mice transgenic for the heavy chain of a pathogenic anti-DNA a
The National Academy of Sciences.