Hypoxic Ischemic Encephalopathy
Mostrando 1-12 de 13 artigos, teses e dissertações.
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1. Hypothyroxinemia in sick term neonates and its risk factors in an extramural neonatal intensive care unit: a prospective cohort study
ABSTRACT Objective: Thyroid functions in the sick newborns may be altered in the first week of life. Transient hypothyroxinemia has been reported in these babies, which could be due to the immaturity of the hypothalamic-pituitary-thyroid axis or to acute illness. We conducted this study to estimate the incidence of hypothyroxinemia and determine its risk fa
Archives of Endocrinology and Metabolism. Publicado em: 2022
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2. Newborns at high risk for brain injury: the role of the amplitude-integrated electroencephalography
Abstract Objective: Amplitude-integrated electroencephalography (aEEG) is a simplified bedside neurophysiology tool that has been implemented in the neonatal intensive care unit and studied in an extensive range of clinical applications in the past decade. This critical review aimed to evaluate a variety of clinical applications of aEEG monitoring in diagno
Jornal de Pediatria. Publicado em: 2022
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3. Brain caspase-3 and intestinal FABP responses in preterm and term rats submitted to birth asphyxia
Neonatal asphyxia can cause irreversible injury of multiple organs resulting in hypoxic-ischemic encephalopathy and necrotizing enterocolitis (NEC). This injury is dependent on time, severity, and gestational age, once the preterm babies need ventilator support. Our aim was to assess the different brain and intestinal effects of ischemia and reperfusion in n
Braz J Med Biol Res. Publicado em: 23/06/2016
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4. Language in Children with Neonatal Hypoxic-Ischemic Encephalopathy
Introduction Neonatal hypoxic-ischemic encephalopathy (NHIE) is a common neurologic injury, and it may compromise the child's language and cognition. Understanding the process of language acquisition becomes possible with concise knowledge about children's global development. Objective The aim of this study was to observe if language acquisition and devel
Int. Arch. Otorhinolaryngol.. Publicado em: 2014-09
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5. Association of NOS3 gene variants and clinical contributors of hypoxic-ischemic encephalopathy
The aim of this study was to analyze the association of different clinical contributors of hypoxic-ischemic encephalopathy with NOS3 gene polymorphisms. A total of 110 children with hypoxic-ischemic encephalopathy and 128 control children were selected for this study. Association of gender, gestational age, birth weight, Apgar score, cranial ultrasonography,
Braz J Med Biol Res. Publicado em: 15/08/2014
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6. S100B protein related neonatal hypoxia / Relação da proteína S100B com a hipóxia neonatal
A participação de marcadores bioquímicos na avaliação de quadros de asfixia neonatal é cada vez mais relevante. A proteína S100B é de particular importância neste campo. O objetivo deste estudo foi procurar destacar a importância da proteína S100B na avaliação de recém-nascidos a termo com quadro de encefalopatia hipóxico-isquêmica, assim com
Publicado em: 2010
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7. Epidemiologia dos óbitos neonatais hospitalares associados à asfixia perinatal no interior do Estado de Alagoas em 2009. / Epidemiology of hospital neonatal deaths associated to perinatal asphyxia in the country side of Alagoas State in 2009.
Objective: To describe epidemiologic frequency and characteristics of intra-hospital neonatal deaths associated to perinatal asphyxia in the country side of Alagoas state in 2009. Method: Prospective cross-seccional study with weekly active search for medical records of mothers and their newborn infants who died before 28 days of age from January 1st to Dece
Publicado em: 2010
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8. Prevalence of perinatal asphyxia and hypoxic-ischemic encephalopathy in term newborns considering two diagnostic criteria and the type of obstetric assistance / Prevalência de asfixia perinatal e encefalopatia hipóxico-isquêmica em recém-nascidos de termo considerando dois critérios diagnósticos e o tipo de assistência obstétrica
INTRODUÇÃO: A asfixia perinatal é uma das principais causa de óbito nos recémnascidos (RN) de termo acima de 2500g no Brasil, sendo também a causa mais importante de encefalopatia e lesão cerebral permanente em crianças. Não existindo ainda um consenso acerca de qual seria o melhor critério para seu diagnóstico. OBJETIVOS: Verificar a prevalência
Publicado em: 2008
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9. Relação da proteína S100B com a hipóxia neontal
A participação de marcadores bioquímicos na avaliação de quadros de asfixia neonatal é cada vez mais relevante. A proteína S100B tem um papel destacado nestas pesquisas. O objetivo deste estudo foi procurar destacar a importância da proteína S100B na avaliação de recém-nascidos a termo com quadros de encefalopatia hipóxico-isquêmica, assim como
Publicado em: 2007
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10. Malformações fetais, defeitos de desenvolvimento e sinais dismorficos em filhos de mães com epilepsia / Fetal malformations, development defects and dysmorphic signs in outcomes of women with epilepsy
Rationale: The majority of women with epilepsy do not experience significant changes during pregnancy; however the persistence of seizures and the effect of antiepileptic drugs (AED) may affect the fetus. These effects include lower intrauterine growth, dysmorphisms, fetal malformation and neuropsicomotor developmental delay. Most studies report that the ris
Publicado em: 2007
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11. Analise da correlação entre o tono muscular, força muscular e as funções motoras em crianças com paralisia cerebral tetraparetica espastica devido a lesões hipoxico- isquemicas
Cerebral palsy (CP) is a clinical picture resulting from nonprogressive aggression of the developing brain, characterized by tonus, muscular strength and motor function alterations. The objective ofthis study was to ana1yzethe tonus and muscular strength interference on motor functions of children with spastic tetraparetic cerebral palsy due to hypoxic ische
Publicado em: 2003
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12. Hypoxic and ischemic hypoxia exacerbate brain injury associated with metabolic encephalopathy in laboratory animals.
Hypoxemia is a major comorbid factor for permanent brain damage in several metabolic encephalopathies. To determine whether hypoxia impairs brain adaptation to hyponatremia, worsening brain edema, we performed in vitro and in vivo studies in cats and rats with hyponatremia plus either ischemic or hypoxic hypoxia. Mortality with hypoxic hypoxia was 0%; with h