Mediodorsal Thalamus
Mostrando 1-6 de 6 artigos, teses e dissertações.
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1. Plasticidade sináptica no córtex pré-frontal induzida por estimulação do tálamo mediodorsal de ratos in vivo: efeitos da modulação colinérgica muscarínica e nicotínica / Prefrontal cortical synaptic plasticity induced by stimulation of the rat mediodorsal thalamus in vivo: effects of cholinergic muscarinic and nicotinic modulation
O núcleo talâmico mediodorsal (Tmd) e o córtex pré-frontal (CPF) comunicam-se mutuamente, formando um circuito envolvido em funções executivas e transtornos psiquiátricos. As funções executivas estão sujeitas aos níveis de alerta gerados pela atividade oscilatória talamocortical, que por sua vez é controlada pela transmissão colinérgica. Possi
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 17/08/2012
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2. Mediodorsal Thalamic Lesions Impair Trace Eyeblink Conditioning in the Rabbit
Rabbits received lesions of the mediodorsal nucleus of the thalamus (MDN) or sham lesions and were subjected to classical eyeblink (EB) and heart rate (HR) conditioning. All animals received trace conditioning, with a .5-sec tone conditioned stimulus, a .5-sec trace period, and a 50-msec periorbital shock unconditioned stimulus. Animals with MDN lesions acqu
Cold Spring Harbor Laboratory Press.
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3. Subnucleus-specific loss of neurons in medial thalamus of schizophrenics
The hypoactivity of dorsolateral prefrontal cortex in schizophrenics is well known. One cause of this hypoactivity may be defective corticocortical or thalamocortical connections. Recent imaging studies of the thalamus suggest reductions in volume of the whole thalamus and reduced activity in the medial group of thalamic nuclei, which may indicate loss of fu
The National Academy of Sciences.
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4. Changes in local cerebral glucose utilization during rewarding brain stimulation.
The quantitative 2-deoxy[14C]glucose method was used to determine local cerebral glucose utilization in unrestrained rats responding (lever-press) for rewarding electrical stimulation to area A10 (ventral tegmental area) and in similarly implanted inactive controls. Self-stimulation was associated with significant increases in metabolic activity, highly circ
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5. Quantitative autoradiography of beta 1- and beta 2-adrenergic receptors in rat brain.
We have used quantitative autoradiography to localize in rat brain beta 1- and beta 2-adrenergic receptors. These receptors were labeled in vitro with 125I-labeled pindolol, an antagonist of beta-adrenergic receptors that binds nonselectively to both beta 1 and beta 2 subtypes. The selective inhibition of 125I-labeled pindolol binding with specific antagonis
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6. Phospholipase C I and II brain isozymes: immunohistochemical localization in neuronal systems in rat brain.
Two distinct inositol phospholipid-specific phospholipase C (PLC; phosphatidylcholine phosphatidohydrolase, EC 3.1.4.3) isozymes, PLC-I and PLC-II, have been purified and characterized from bovine brain. Monoclonal antibodies that distinguish between these isozymes are used in the present study to map isozyme distribution in the rat brain with immunohistoche