Oral Epithelial Dysplasias
Mostrando 1-4 de 4 artigos, teses e dissertações.
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1. Oral potentially malignant disorders in a large dental population
Objectives Oral cancer (OC) may be preceded by clinically evident oral potentially malignant disorders (OPMDs). Oral carcinogenesis is a multistep process that begins as epithelial hyperplasia and progresses to oral epithelial dysplasia and finally to fully malignant phenotypes. The aim of our study was to estimate the prevalence of OPMDs in a large popula
J. Appl. Oral Sci.. Publicado em: 2014-12
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2. Expression of the cell cycle regulation proteins p53 and p21WAF1 in different types of non-dysplastic leukoplakias
OBJECTIVES: The aim of this study was to analyze the immunolabeling of two cell cycle protein regulators, p53 and p21WAF1, in non-dysplastic leukoplakias with different epithelial alterations: acanthosis, hyperkeratosis and acanthosis combined with hyperkeratosis, and compare them with dysplastic leukoplakias. MATERIAL AND METHODS: This was a prospective coh
Journal of Applied Oral Science. Publicado em: 2012-06
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3. Expressão imuno-histoquímica do cd8, foxp3, Tgf β, tnf α e nf-кb em displasias epiteliais e Carcinomas epidermóides orais
A Displasia Epitelial Oral (DEO) é a lesão que precede ou co-existe com o Carcinoma Epidermóide Oral (CEO), apresentando alterações moleculares e/ou histológicas semelhantes. As divergências sobre o potencial de malignização das DEO e o papel da inflamação nestes processos têm dificultado o diagnóstico precoce e a avaliação da agressividade do
Publicado em: 2009
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4. Skp2 is oncogenic and overexpressed in human cancers
Skp2 is a member of the F-box family of substrate-recognition subunits of SCF ubiquitin–protein ligase complexes that has been implicated in the ubiquitin-mediated degradation of several key regulators of mammalian G1 progression, including the cyclin-dependent kinase inhibitor p27, a dosage-dependent tumor suppressor protein. In this study, we examin
The National Academy of Sciences.