Palatal Shelves
Mostrando 1-12 de 14 artigos, teses e dissertações.
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1. Avaliação do tamanho das lâminas palatinas no resultado do crescimento dos arcos dentários e relação interarcos na dentadura decídua ou mista precoce / Evaluation of the size of palatal shelves on the outcome of dental arch growth and interarch relationship in the deciduous or early mixed dentition.
Trata-se de um estudo retrospectivo, com o objetivo de avaliar a correlação entre o tamanho das lâminas palatinas e índice oclusal em pacientes com fissura unilateral completa de lábio e palato. Foram avaliados modelos de estudo de 339 pacientes que foram moldados em três fases distintas: pré-queiloplastia (fase1) pré-palatoplastia (fase 2) e dentadu
IBICT - Instituto Brasileiro de Informação em Ciência e Tecnologia. Publicado em: 19/10/2011
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2. Expressão temporal e espacial de RECK, metaloproteinase-2 e metaloproteinase-9 durante a palatogenese em camundongos / Temporal and spatial expression of RECK, metalloproteinase-2 and metalloproteinase-9 during palatogenesis in mice
The oral clefts show a great influence in the size of their hosts. They have, since birthy, difficulties in alimentation, problems in the maxilar bones, in dental arch, in speech, and in some cases, in hearing. In addition, the hosts and their families have serious psychological problems due to the esthetic lenght caused by anomalia. With no doubt, the forma
Publicado em: 2007
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3. Indirect modulation of Shh signaling by Dlx5 affects the oral-nasal patterning of palate and rescues cleft palate in Msx1-null mice
Cleft palate represents one of the most common congenital birth defects in human. During embryonic development, palatal shelves display oronasal (O-N) and anteroposterior polarity before the onset of fusion, but how the O-N pattern is established and how it relates to the expansion and fusion of the palatal shelves are unknown. Here we address these question
Company of Biologists.
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4. Experimental induction of premature movement of rat palatal shelves in vivo.
In order to enhance further knowledge of palatal shelf movement and the factors involved in palate closure, a method was developed for prematurely elevating palatal shelves in utero. Approximately 7 hours before expected shelf elevation, pregnant Sprague-Dawley rats were laparotomized and two medially directed squeezes were applied to the face of some of the
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5. TGF-β3-induced Palatogenesis Requires Matrix Metalloproteinases
Cleft lip and palate syndromes are among the most common congenital malformations in humans. Mammalian palatogenesis is a complex process involving highly regulated interactions between epithelial and mesenchymal cells of the palate to permit correct positioning of the palatal shelves, the remodeling of the extracellular matrix (ECM), and subsequent fus
The American Society for Cell Biology.
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6. Isolated cleft palate in mice with a targeted mutation of the LIM homeobox gene Lhx8
Formation of the mammalian secondary palate is a highly regulated and complex process whose impairment often results in cleft palate, a common birth defect in both humans and animals. Loss-of-function analysis has linked a growing number of genes to this process. Here we report that Lhx8, a recently identified LIM homeobox gene, is expressed in the mesenchym
The National Academy of Sciences.
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7. Palatal shelf elevation in the Wistar rat fetus.
Palatogenesis in the Wistar rat fetus was studied macroscopically, microscopically, ultrastructurally and experimentally between days 13 and 19. The developmental ages of the fetuses were calculated from the smear age of the litter adjusted for individual variations in crown-rump lengths. Palatal shelf elevation occurs at day 16.4 +/- 0.1. Experimentally ind
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8. Epithelial cell death and cyclic AMP increase during palatal development.
Cyclic AMP levels increase abruptly in the secondary palatal shelf (mesenchyme plus a two- to three-cell-layered epithelium) between day 15 and 16 of gestation in the rat embryo. The addition of dibutyryl cyclic AMP to the media in which immature palatal shelves (day 14) are cultured causes a precocious decrease in DNA synthesis, but increased glycoprotein s
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9. The teratogenic effects of 5-fluoro-2-desoxyuridine (F.U.D.R.) on the Wistar rat fetus with particular reference to cleft palate.
The teratogenic effects of 5-fluoro-2-desoxyuridine on Wistar rat fetuses were studied macroscopically, microscopically and ultrastructurally. In no case did palatal shelf elevation occur, and a palatal shelf elevating force could not be demonstrated in freshly-removed fetuses. The shelves, like the connective tissues generally, showed clear evidence of depr
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10. Glucocorticoid-induced phospholipase A2-inhibitory proteins mediate glucocorticoid teratogenicity in vitro.
Dexamethasone induces the synthesis of a phospholipase A2-inhibitory protein (PLIP) of molecular weight approximately equal to 55,000 from calf thymus and PLIPs of molecular weights 55,000, 40,000, 28,000, and 15,000 from A/J mouse thymus and from 12-day embryonic B10. A mouse palates. Sufficient quantities of calf thymus PLIP and of the 15,000 molecular wei
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11. Cleft lip with or without cleft palate: implication of the heavy chain of non‐muscle myosin IIA
Non‐syndromic cleft lip with or without palate (CL/P) is one of the most common malformations among live births, but most of the genetic components and environmental factors involved remain to be identified. Among the different causes, MYH9, the gene encoding for the heavy chain of non‐muscle myosin IIA, was considered a potential candidate, because it w
BMJ Group.
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12. Cleft palate development in hamster embryos following triamcinolone treatment.
Development of the palate was studied in normal and triamcionolone-treated hamster fetuses. The results demonstrated that normal palatogenesis was completed between days 12 and 13 of gestation. Following triamcinolone treatment the reorientation of the palatal shelves was delayed before there was any general retardation of fetal growth (as indicated by crown