Paraparesis Tropical Spastic Therapy
Mostrando 1-5 de 5 artigos, teses e dissertações.
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1. Using the International Classification of Functioning, Disability and Health as a tool for analysis of the effect of physical therapy on spasticity in HAM/TSP patients
INTRODUCTION: This study aimed to evaluate spasticity in human T-lymphotropic virus type 1-associated myelopathy/tropical spastic paraparesis (HAM/TSP) patients before and after physical therapy using the International Classification of Functioning, Disability and Health (ICF). METHODS: Nine subjects underwent physical therapy. Spasticity was evaluated usin
Rev. Soc. Bras. Med. Trop.. Publicado em: 2015-04
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2. The use of botulinum toxin type A in the treatment of HTLV-1-associated overactive bladder refractory to conventional therapy
Urinary symptoms occur in 19% of human T-cell lymphotropic virus type 1 (HTLV-1)-infected patients who do not fulfill criteria for HTLV-1-associated myelopathy/tropical spastic paraparesis (HAM/TSP) and in almost 100% of HAM/TSP patients. Few studies have evaluated therapies for overactive bladder (OAB) caused by HTLV-1 infection. This case report describes
Rev. Soc. Bras. Med. Trop.. Publicado em: 2014-08
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3. "Ensaio clínico controlado randomizado aberto com metil-prednisolona em portadores de mielopatia associada ao HTLV-1 paraparesia espástica tropical" / Clinical controlled randomized open trial with methylprednisolone in myelopathy associated to HTLV-1 / tropical spastic paraparesis
Estudamos a avaliação clínica de longo termo da resposta de pacientes com diagnóstico precoce de HAM/TSP ao tratamento com metil-prednisolona. Objetivamos pesquisar em relação ao tempo de evolução e apresentação da doença a eficácia deste tratamento. Realizamos o acompanhamento dos pacientes utilizando como parâmetros as escalas de critérios cl
Publicado em: 2004
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4. Methylprednisolone therapy in tropical spastic paraparesis.
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5. Valproate activates bovine leukemia virus gene expression, triggers apoptosis, and induces leukemia/lymphoma regression in vivo
Leukemogenic viruses like human T-lymphotropic virus and bovine leukemia virus (BLV) presumably persist in the host partly by latent integration of the provirus in a fraction of infected cells, leading to accumulative increase in the outgrowth of transformed cells. Furthermore, viral infection also correlates with a blockade of the apoptotic mechanisms conco
National Academy of Sciences.